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Disease Info Card
Ectopia Cordis
Information about Ectopia Cordis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Ectopia Cordis
Most recent studies have shown that Ectopia Cordis shares some biological mechanisms with cardiac-arrhythmia, cardiovascular-abnormalities, cattle-diseases, congenital-abnormality, congenital-ectopia, congenital-exomphalos, congenital-heart-defects, congenital-heart-disease, congenital-omphalocele, congenital-sternal-cleft, cytogenetic-abnormality, fetal-diseases, gastroschisis, heart-diseases, heart-septal-defects, hernia, pentalogy-of-cantrell, tetralogy-of-fallot, ventricular-septal-defects.
Among the many pathways, these few ones have gauged particular interests from scientists studying Ectopia Cordis, and have been seen in publications frequently: Angiogenesis, Cell Cycle, Cell Differentiation, Cell Migration, Cell Proliferation, Ductus Arteriosus Closure, Endothelial Cell Proliferation, Heart Development, Heart Formation, Heart Induction, Insemination, Localization, Membrane Depolarization, Neural Tube Closure, Pathogenesis, Signal Transmission, T Cell Differentiation, Transposition, Tube Closure, Tube Formation
Quite a number of genes have been found to play important roles in Ectopia Cordis, such as ACHE, AFP, BCHE, CD4, CD40, CD40LG, CD80, CD8A, CER1, DKK1, HHEX, NR4A3, NT5E, NTM, NTS, QPCT, TCF7L1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.