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Disease Info Card
Dysfibrinogenemia
Information about Dysfibrinogenemia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Dysfibrinogenemia
Most recent studies have shown that Dysfibrinogenemia shares some biological mechanisms with afibrinogenemia, antithrombin-iii-deficiency, bleeding-tendency, blood-coagulation-disorders, congenital-dysfibrinogenaemia, hemorrhage, hemorrhagic-disorders, hypofibrinogenemia, liver-cirrhosis, liver-diseases, lupus-anticoagulant-disorder, lupus-erythematosus-systemic, pregnancy-complications-hematologic, protein-c-deficiency, protein-s-deficiency, thromboembolism, thrombophilia, thrombosis, venous-thrombosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Dysfibrinogenemia, and have been seen in publications frequently: Blood Coagulation, Cell Adhesion, Cell Migration, Coagulation, Detection Of Activity, Erythrocyte Aggregation, Fibrinolysis, Hemostasis, Localization, Muscle Cell Migration, Pathogenesis, Plasminogen Activation, Platelet Activation, Platelet Aggregation, Proteolysis, Regulation Of Blood Coagulation, Secretion, Translation, Tube Formation, Wound Healing
Quite a number of genes have been found to play important roles in Dysfibrinogenemia, such as ALB, APC, F2, F5, FGA, FGB, FGG, PLAT, PLG, SERPINC1, SERPINE1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.