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Disease Info Card
Afibrinogenemia
Information about Afibrinogenemia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Afibrinogenemia
Most recent studies have shown that Afibrinogenemia shares some biological mechanisms with bleeding-tendency, blood-coagulation-disorders, blood-platelet-disorders, disseminated-intravascular-coagulation, dysfibrinogenemia, fetal-death, hemophilia-a, hemorrhage, hemorrhagic-disorders, hereditary-factor-i-deficiency-disease, hypofibrinogenemia, inherited-factor-ii-deficiency, leukemia, postpartum-hemorrhage, pregnancy-complications, pregnancy-complications-hematologic, thrombosis, uterine-hemorrhage, von-willebrand-disease.
Among the many pathways, these few ones have gauged particular interests from scientists studying Afibrinogenemia, and have been seen in publications frequently: Blood Coagulation, Cell Migration, Cell Proliferation, Coagulation, Complement Activation, Erythrocyte Aggregation, Fibrinolysis, Hemostasis, Hypersensitivity, Menstruation, Mrna Splicing, Ovulation, Pathogenesis, Plasminogen Activation, Platelet Activation, Platelet Aggregation, Proteolysis, Secretion, Translation, Wound Healing
Quite a number of genes have been found to play important roles in Afibrinogenemia, such as ALB, C3, CFH, CFI, F2, F8, FGA, FGB, FGG, FN1, PLAT, PLG, SERPINC1, VWF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.