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- Table of Contents
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Disease Info Card
Lupus Anticoagulant Disorder
Information about Lupus Anticoagulant Disorder: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Lupus Anticoagulant Disorder
Most recent studies have shown that Lupus Anticoagulant Disorder shares some biological mechanisms with acute-promyelocytic-leukemia, antiphospholipid-syndrome, arterial-thrombosis, autoimmune-diseases, autoimmune-reaction, blood-coagulation-disorders, cerebrovascular-accident, deep-vein-thrombosis, hemorrhage, hypertensive-disease, infarction, lupus-erythematosus-systemic, polyendocrinopathies-autoimmune, pregnancy-complications, thromboembolism, thrombophilia, thrombosis, venous-thrombosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Lupus Anticoagulant Disorder, and have been seen in publications frequently: B Cell Activation, Blood Coagulation, Cell Activation, Coagulation, Complement Activation, Endothelial Cell Activation, Excretion, Fertilization, Fibrinolysis, Glomerular Filtration, Hemostasis, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Pathogenesis, Platelet Activation, Platelet Aggregation, Proteolysis, Secretion
Quite a number of genes have been found to play important roles in Lupus Anticoagulant Disorder, such as ANXA5, APC, C3, F10, F2, F5, GNPDA1, GPI, KLK3, LCT, MTHFR, PLG, PROC, SERPINC1, SH2B2, VWF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.