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Disease Info Card
Antithrombin Iii Deficiency
Information about Antithrombin Iii Deficiency: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Antithrombin Iii Deficiency
Most recent studies have shown that Antithrombin Iii Deficiency shares some biological mechanisms with activated-protein-c-resistance, antiphospholipid-syndrome, blood-coagulation-disorders, deep-vein-thrombosis, disseminated-intravascular-coagulation, embolism, hemorrhage, infarction, lupus-erythematosus-systemic, pregnancy-complications-hematologic, protein-c-deficiency, protein-s-deficiency, pulmonary-embolism, thromboembolism, thrombophilia, thrombophlebitis, thrombosis, venous-thromboembolism, venous-thrombosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Antithrombin Iii Deficiency, and have been seen in publications frequently: Blood Coagulation, Coagulation, Excretion, Fibrinolysis, Glycosylation, Hemostasis, Inflammatory Response, Localization, Mating, Ovulation, Parturition, Pathogenesis, Platelet Aggregation, Proteolysis, Regulation Of Blood Coagulation, Regulation Of Coagulation, Regulation Of Hemostasis, Response To Heparin, Secretion, Translation
Quite a number of genes have been found to play important roles in Antithrombin Iii Deficiency, such as APC, ASRGL1, ATAT1, F10, F2, F5, LPA, MTHFR, PLG, PROC, SERPINC1, SLC25A10, TAT. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.