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Disease Info Card
Disuse Muscle Atrophy
Information about Disuse Muscle Atrophy: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Disuse Muscle Atrophy
Most recent studies have shown that Disuse Muscle Atrophy shares some biological mechanisms with arthritis, atrophy, degenerative-polyarthritis, disuse-osteoporosis, flexed-fetal-attitude, fracture, hypertrophy, inflammation, muscle-fiber-atrophy, muscle-twitch, muscle-weakness, muscular-atrophy, muscular-disorders-atrophic, muscular-dystrophy, myopathy, neuromuscular-diseases, osteopenia, pain, weakness.
Among the many pathways, these few ones have gauged particular interests from scientists studying Disuse Muscle Atrophy, and have been seen in publications frequently: Aging, Angiogenesis, Autophagy, Bone Remodeling, Bone Resorption, Excretion, Flight, Glycolysis, Hibernation, Innervation, Muscle Adaptation, Muscle Atrophy, Muscle Hypertrophy, Pathogenesis, Proteolysis, Reflex, Regeneration, Secretion, Skeletal Muscle Atrophy, Transport
Quite a number of genes have been found to play important roles in Disuse Muscle Atrophy, such as AKT1, CASP3, CAT, CRAT, CS, CSH1, DMD, ERCC8, FBXO32, HSPA4, HSPA9, IGF1, INS, LIPG, MSTN, MTOR, NFKB1, TNF, TRIM63. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.