Disease Info Card

Disseminated Candidiasis

Information about Disseminated Candidiasis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Disseminated Candidiasis

Most recent studies have shown that Disseminated Candidiasis shares some biological mechanisms with abscess, aspergillosis, candidemia, candidiasis, candidiasis-invasive, cell-invasion, cross-infection, decreased-immunologic-activity-[pe], fungemia, immunologic-deficiency-syndromes, infection-by-candida-albicans, infection-by-cryptococcus-neoformans, infective-disorder, leukemia, malignant-neoplasms, mycoses, neoplasms, opportunistic-infections, oral-candidiasis, systemic-infection.

Among the many pathways, these few ones have gauged particular interests from scientists studying Disseminated Candidiasis, and have been seen in publications frequently: Biofilm Formation, Coagulation, Complement Activation, Cytokine Production, Drug Resistance, Fermentation, Filamentous Growth, Host-pathogen Interaction, Hypersensitivity, Hyphal Growth, Immune Response, Inflammatory Response, Innate Immune Response, Localization, Pathogenesis, Phagocytosis, Secretion, Transport, Tube Formation, Virulence

Quite a number of genes have been found to play important roles in Disseminated Candidiasis, such as C3, C5, CSF2, CSF3, ENO1, GNAI1, HSP90AA1, IFNG, IL10, IL2, IL4, IL6, LAMC2, MAP3K10, MPST, PLB1, SOD1, TLR2, TLR4, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.