Disease Info Card

Infection By Candida Albicans

Information about Infection By Candida Albicans: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Infection By Candida Albicans

Most recent studies have shown that Infection By Candida Albicans shares some biological mechanisms with abscess, agranulocytosis, candidiasis, candidiasis-of-vagina, candidiasis-vulvovaginal, cutaneous-candidiasis, decreased-immunologic-activity-[pe], delayed-hypersensitivity, disseminated-candidiasis, immunologic-deficiency-syndromes, infective-disorder, inflammation, lung-diseases-fungal, malignant-neoplasms, mycoses, neoplasms, opportunistic-infections, oral-candidiasis, pregnancy-complications-infectious, systemic-infection.

Among the many pathways, these few ones have gauged particular interests from scientists studying Infection By Candida Albicans, and have been seen in publications frequently: Cell Activation, Cell Adhesion, Cell Proliferation, Coagulation, Cytokine Production, Cytokine Secretion, Drug Resistance, Humoral Immune Response, Hypersensitivity, Hyphal Growth, Immune Response, Inflammatory Response, Innate Immune Response, Lactation, Macrophage Activation, Opsonization, Pathogenesis, Phagocytosis, Secretion, Virulence

Quite a number of genes have been found to play important roles in Infection By Candida Albicans, such as C5, CD4, CD86, CSF2, CSF3, ICAM1, IFNG, IL10, IL17A, IL18, IL2, IL4, IL6, MPO, TLR2, TLR4, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.