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Disease Info Card
Dental Fluorosis, Acquired
Information about Dental Fluorosis, Acquired: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Dental Fluorosis, Acquired
Most recent studies have shown that Dental Fluorosis, Acquired shares some biological mechanisms with bone-diseases, cattle-diseases, dental-caries, dental-enamel-hypoplasia, dental-plaque, endemic-diseases, fracture, gingivitis, hypoplasia, malocclusion, metabolic-bone-disorder, mottled-teeth-congenital, osteomalacia, osteoporosis, osteosclerosis, pain, periodontal-diseases, poisoning, tooth-diseases.
Among the many pathways, these few ones have gauged particular interests from scientists studying Dental Fluorosis, Acquired, and have been seen in publications frequently: Aging, Amelogenesis, Blood Coagulation, Bone Mineralization, Bone Remodeling, Bone Resorption, Cell Proliferation, Coagulation, Enamel Mineralization, Excretion, Lactation, Mastication, Ossification, Pathogenesis, Pigmentation, Reflex, Regeneration, Secretion, Tooth Eruption, Transport
Quite a number of genes have been found to play important roles in Dental Fluorosis, Acquired, such as ALB, ARSH, BEST1, BGLAP, CAT, CFI, DMD, F3, GRB2, MMP20, MMP25, PTH, PTRH1, RANGAP1, SLC17A5, SOD1, SS18L1, TF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.