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Disease Info Card
Amelogenesis Imperfecta
Information about Amelogenesis Imperfecta: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Amelogenesis Imperfecta
Most recent studies have shown that Amelogenesis Imperfecta shares some biological mechanisms with anterior-open-bite, congenital-abnormality, dental-caries, dental-enamel-hypoplasia, dental-fluorosis-acquired, dentin-dysplasia, dentin-sensitivity, dentinogenesis-imperfecta, developmental-absence-of-tooth, dysplasia, exanthema, hereditary-diseases, hypodontia, hypoplasia, impacted-tooth, malocclusion, open-bite, tooth-abnormalities.
Among the many pathways, these few ones have gauged particular interests from scientists studying Amelogenesis Imperfecta, and have been seen in publications frequently: Amelogenesis, Bone Resorption, Cell Adhesion, Cell Differentiation, Cell Proliferation, Dentinogenesis, Enamel Mineralization, Hypersensitivity, Localization, Mating, Odontogenesis, Pathogenesis, Pigmentation, Protein Secretion, Proteolysis, Root Development, Secretion, Tooth Eruption, Translation, Transport
Quite a number of genes have been found to play important roles in Amelogenesis Imperfecta, such as ACP5, ALB, AMBN, AMELX, AMTN, CNNM4, DLX3, DSPP, ENAM, FAM83H, KLK4, MMP20, MMP25, TDO2, TDRD7, TUFT1, WDR72. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.