Disease Info Card

Complement Deficiency

Information about Complement Deficiency: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Complement Deficiency

Most recent studies have shown that Complement Deficiency shares some biological mechanisms with angioedema, arthritis, autoimmune-diseases, autoimmune-reaction, autoimmunity, bacterial-infections, glomerulonephritis, hemolysis-(disorder), immune-complex-diseases, immunologic-deficiency-syndromes, infective-disorder, inflammation, lupus-erythematosus-systemic, malnutrition, meningitis, meningitis-bacterial, meningococcal-infections, meningococcal-meningitis, pneumonia, systemic-infection.

Among the many pathways, these few ones have gauged particular interests from scientists studying Complement Deficiency, and have been seen in publications frequently: Antibody-dependent Cellular Cytotoxicity, Apoptotic Cell Clearance, Cell Activation, Cell Adhesion, Coagulation, Complement Activation, Humoral Immune Response, Immune Complex Clearance, Immune Response, Inflammatory Response, Localization, Macrophage Activation, Opsonization, Pathogenesis, Phagocytosis, Pigmentation, Proteolysis, Secretion, T Cell Activation, Virulence

Quite a number of genes have been found to play important roles in Complement Deficiency, such as AKR1C1, ARHGAP4, C2, C3, C4A, C4B, C5, C6, C7, C9, CFH, CFP, CR1, CXCL10, HNRNPC, PFDN4, PSMA7. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.