Disease Info Card

Chorioretinal Atrophy

Information about Chorioretinal Atrophy: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Chorioretinal Atrophy

Most recent studies have shown that Chorioretinal Atrophy shares some biological mechanisms with age-related-macular-degeneration, atrophy, blind-vision, choroid-diseases, choroidal-neovascularization, disorder-of-eye, dystrophy, glaucoma, gyrate-atrophy, macule, myopia, myopia-degenerative, optic-atrophy, pathologic-neovascularization, retinal-degeneration, retinal-detachment, retinal-diseases, retinitis-pigmentosa, severe-myopia, visual-impairment.

Among the many pathways, these few ones have gauged particular interests from scientists studying Chorioretinal Atrophy, and have been seen in publications frequently: Aging, Blood Circulation, Cell Proliferation, Coagulation, Enucleation, Excretion, Hatching, Immune Response, Localization, Neurotransmitter Uptake, Pathogenesis, Phototransduction, Pigment Accumulation, Pigmentation, Reflex, Regeneration, Reverse Transcription, Segmentation, Translation, Transport

Quite a number of genes have been found to play important roles in Chorioretinal Atrophy, such as ABCA4, AIPL1, CHM, CRX, ERG, FANCA, FXN, KCNH2, KCNMA1, MPZ, OAT, PLXNA2, PRPH, RPE, TEAD1, VCAN, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.