Disease Info Card

Charcot-marie-tooth Disease, Type 4c

Information about Charcot-marie-tooth Disease, Type 4c: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Charcot-marie-tooth Disease, Type 4c

Most recent studies have shown that Charcot-marie-tooth Disease, Type 4c shares some biological mechanisms with atrophy, charcot-marie-tooth-disease, cmt4b1, congenital-abnormality, curvature-of-spine, cytogenetic-abnormality, foot-deformities, hereditary-motor-and-sensory-neuropathies, inherited-neuropathy, muscular-atrophy, neuromuscular-diseases, peripheral-neuropathy, polyneuropathy, scoliosis-unspecified, sensory-neuropathy, weakness.

Among the many pathways, these few ones have gauged particular interests from scientists studying Charcot-marie-tooth Disease, Type 4c, and have been seen in publications frequently: Cell Adhesion, Endocytic Recycling, Endosome Localization, Localization, Myelination, Pathogenesis

Quite a number of genes have been found to play important roles in Charcot-marie-tooth Disease, Type 4c, such as AKR1B1, AR, AREG, EGR2, FDXR, GDAP1, KIF1B, KIF20A, MFN2, MPZ, MTMR2, MTSS1, MYCN, NDRG1, SH3TC2. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.

Charcot-marie-tooth Disease, Type 4c Related Genes

click to see detail information for each gene

AKR1B1 AR AREG
EGR2 FDXR GDAP1
KIF1B KIF20A MFN2
MPZ MTMR2 MTSS1
MYCN NDRG1 SH3TC2

Pathways Related to Charcot-marie-tooth Disease, Type 4c

This information is being compiled and will come in a future update

Cell Adhesion Endocytic Recycling Endosome Localization
Localization Myelination Pathogenesis