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Disease Info Card
Cardiopulmonary Arrest
Information about Cardiopulmonary Arrest: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Cardiopulmonary Arrest
Most recent studies have shown that Cardiopulmonary Arrest shares some biological mechanisms with apnea, brain-injuries, cardiac-arrest, cardiac-arrhythmia, cardiac-fibrillation, comatose, edema, embolism, encephalopathies, hemorrhage, hypotension-adverse-event, hypoxia, infarction, ischemia, myocardial-infarction, pain, pneumonia, ventricular-fibrillation.
Among the many pathways, these few ones have gauged particular interests from scientists studying Cardiopulmonary Arrest, and have been seen in publications frequently: Anaphylaxis, Cardiac Conduction, Cell Death, Coagulation, Cognition, Diuresis, Donor Selection, Excretion, Flight, Glycolysis, Hypersensitivity, Localization, Oxygen Transport, Pathogenesis, Reflex, Response To Hypoxia, Response To Pain, Swimming, Transport, Vasoconstriction
Quite a number of genes have been found to play important roles in Cardiopulmonary Arrest, such as AVP, C2, CA1, CAT, CPA1, CPB1, CRAT, CSF2, GLI3, GLYAT, IGFALS, KIF7, LAMC2, MET, NDUFB6, POR, RNMT, SLC17A5, SLTM, SOD1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.