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Disease Info Card
Borrelia Infections
Information about Borrelia Infections: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Borrelia Infections
Most recent studies have shown that Borrelia Infections shares some biological mechanisms with acrodermatitis-atrophicans-chronica, arthritis, communicable-diseases, encephalitis, erythema, erythema-chronicum-migrans, glossitis-benign-migratory, infective-disorder, lyme-arthritis, lyme-disease, lyme-neuroborreliosis, meningitis, nervousness, relapsing-fever, tick-bite-(disorder), tick-borne-diseases, tick-borne-encephalitis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Borrelia Infections, and have been seen in publications frequently: Antigenic Variation, Chemotaxis, Coagulation, Complement Activation, Cytokine Production, Dna Amplification, Humoral Immune Response, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Lymphocyte Proliferation, Pathogenesis, Phagocytosis, Reflex, Response To Antibiotic, Secretion, Sensitization, Tropism, Virulence
Quite a number of genes have been found to play important roles in Borrelia Infections, such as ATP6V0D1, C3, C6, CDK5R2, CFH, CSF2, CUX1, EPHA5, IFNG, IL10, IL4, IL6, JUN, LAMC2, MAPK1, PMEL, PSMA7, TLR2, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.