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Disease Info Card
Bone Demineralization, Pathologic
Information about Bone Demineralization, Pathologic: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Bone Demineralization, Pathologic
Most recent studies have shown that Bone Demineralization, Pathologic shares some biological mechanisms with arthritis, atrophy, bone-diseases, decalcification, fracture, hypokinesia, kidney-diseases, kidney-failure-chronic, malignant-neoplasms, metabolic-bone-disorder, motion-sickness, muscular-atrophy, osteomalacia, osteopenia, osteoporosis, pain, rheumatoid-arthritis, space-motion-sickness.
Among the many pathways, these few ones have gauged particular interests from scientists studying Bone Demineralization, Pathologic, and have been seen in publications frequently: Aging, Bone Development, Bone Mineralization, Bone Remodeling, Bone Resorption, Cell Adhesion, Eating Behavior, Excretion, Flight, Hibernation, Lactation, Menarche, Menopause, Muscle Atrophy, Ossification, Osteoblast Differentiation, Osteoclast Differentiation, Pathogenesis, Secretion, Transport
Quite a number of genes have been found to play important roles in Bone Demineralization, Pathologic, such as BEST1, BGLAP, CALCA, CEACAM1, DMD, FGF23, IGF1, PHEX, PTH, PTRH1, RANGAP1, RAPGEF5, SLA2, SLC17A5, SLC9A3R1, TNFRSF11A, TNFRSF11B, TNFSF11. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.