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Disease Info Card
Bladder Exstrophy
Information about Bladder Exstrophy: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Bladder Exstrophy
Most recent studies have shown that Bladder Exstrophy shares some biological mechanisms with adenocarcinoma, anus-imperforate, bladder-neoplasm, blepharoptosis, cloacal-exstrophy, congenital-abnormality, congenital-exomphalos, epispadias, gastroesophageal-reflux-disease, hydronephrosis, neurogenic-urinary-bladder, pathologic-fistula, stenosis, urethral-diseases, urinary-bladder-diseases, urinary-incontinence, urinary-tract-infection, vesico-ureteral-reflux.
Among the many pathways, these few ones have gauged particular interests from scientists studying Bladder Exstrophy, and have been seen in publications frequently: Anaphylaxis, Bone Mineralization, Cell Migration, Cell Proliferation, Dehiscence, Fertilization, Glomerular Filtration, Hypersensitivity, Innervation, Keratinization, Micturition, Neural Tube Closure, Pathogenesis, Reflex, Regeneration, Segmentation, Sensitization, System Development, Transport, Transposition
Quite a number of genes have been found to play important roles in Bladder Exstrophy, such as ACE, AFP, BEST1, CIC, CKAP4, DMD, EGF, FGF10, GAST, GNAI1, GPSM2, KLK3, MYH14, NDUFB6, RAPGEF5, RPE65, SS18L1, TP63, UVRAG. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.