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Disease Info Card
Renal Tubular Necrosis
Information about Renal Tubular Necrosis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Renal Tubular Necrosis
Most recent studies have shown that Renal Tubular Necrosis shares some biological mechanisms with acute-kidney-injury, edema, fibrosis, glomerulonephritis, hemorrhage, inflammation, injury-to-kidney, ischemia, kidney-diseases, kidney-failure, kidney-failure-acute, kidney-failure-chronic, kidney-tubular-necrosis-acute, nephritis, nephritis-interstitial, oliguria, proteinuria-of-undiagnosed-cause, reperfusion-injury, thrombosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Renal Tubular Necrosis, and have been seen in publications frequently: Aging, Anion Transport, Cell Adhesion, Cell Death, Cell Proliferation, Coagulation, Complement Activation, Diuresis, Excretion, Glomerular Filtration, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Organic Anion Transport, Pathogenesis, Regeneration, Secretion, Transport, Vasoconstriction
Quite a number of genes have been found to play important roles in Renal Tubular Necrosis, such as AKR1B1, ALB, AR, AREG, C3, CAT, CD55, CDKN2A, EGF, FDXR, IL6, MB, MPO, NAGLU, NBAS, PCNA, RAPGEF5, SCN7A, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.