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Disease Info Card
Acute Interstitial Pneumonia
Information about Acute Interstitial Pneumonia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Acute Interstitial Pneumonia
Most recent studies have shown that Acute Interstitial Pneumonia shares some biological mechanisms with abdominal-pain, acute-intermittent-porphyria, autoimmune-diseases, autoimmune-reaction, disorders-of-porphyrin-metabolism, edema, fibrosis, icterus, lung-diseases, lung-diseases-interstitial, malignant-neoplasm-of-pancreas, malignant-neoplasms, neoplasms, pain, pancreatitis, pancreatitis-chronic, pneumonia, pneumonia-interstitial, pulmonary-fibrosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Acute Interstitial Pneumonia, and have been seen in publications frequently: Aging, Cell Cycle, Cell Death, Cell Division, Cell Proliferation, Cholesterol Esterification, Excretion, Hypersensitivity, Immune Response, Insulin Secretion, Localization, Menopause, Neuroprotection, Pathogenesis, Quorum Sensing, Regeneration, Secretion, Translation, Transport, Virulence
Quite a number of genes have been found to play important roles in Acute Interstitial Pneumonia, such as AHR, AIP, AURKAIP1, CALM1, CALM2, CALM3, CDKN1B, CPOX, CRP, FECH, G4, HMBS, INS, MEN1, PAM, PPOX, TG, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.