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Disease Info Card
Acquired Phimosis
Information about Acquired Phimosis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Acquired Phimosis
Most recent studies have shown that Acquired Phimosis shares some biological mechanisms with balanitis, balanitis-xerotica-obliterans, carcinoma, cryptorchidism, hemorrhage, hypospadias, inflammation, lichen-disease, lichen-sclerosus-et-atrophicus, malignant-neoplasm-of-penis, malignant-neoplasms, malignant-squamous-cell-neoplasm, neoplasms, penile-diseases, penile-neoplasms, stenosis, tissue-adhesions, urethral-diseases, urinary-tract-infection.
Among the many pathways, these few ones have gauged particular interests from scientists studying Acquired Phimosis, and have been seen in publications frequently: Blood Coagulation, Cell Differentiation, Cell Proliferation, Circadian Rhythm, Coagulation, Dehiscence, Epithelial Cell Proliferation, Fertilization, Fibrinolysis, Hemostasis, Immune Response, Keratinization, Localization, Menstruation, Micturition, Pathogenesis, Pigmentation, Secretion, Transposition, Wound Healing
Quite a number of genes have been found to play important roles in Acquired Phimosis, such as ACAT1, AR, ARSA, BRD2, C2, CALCR, CDKN2A, CES2, CRAT, DBP, FLAD1, FRZB, GRAP, HSD17B4, PGR, RANGAP1, SERPINB3, SLC17A5, SLC25A5. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.