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Disease Info Card
Wolf-hirschhorn Syndrome
Information about Wolf-hirschhorn Syndrome: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Wolf-hirschhorn Syndrome
Most recent studies have shown that Wolf-hirschhorn Syndrome shares some biological mechanisms with chromosomal-translocation, cleft-lip, congenital-abnormality, craniofacial-abnormalities, cytogenetic-abnormality, developmental-delay-(disorder), developmental-disabilities, epilepsy, fetal-growth-retardation, growth-disorders, growth-retardation, loss-of-chromosome-4, microcephaly, monosomy, multiple-congenital-anomalies, muscle-hypotonia, orbital-separation-excessive, trisomy.
Among the many pathways, these few ones have gauged particular interests from scientists studying Wolf-hirschhorn Syndrome, and have been seen in publications frequently: Bone Maturation, Brain Development, Cell Cycle, Cell Cycle Arrest, Cell Death, Cell Growth, Cell Migration, Chromatin Organization, Histone Modification, Hypersensitivity, Localization, Meiosis, Metaphase, Methylation, Mitosis, Myelination, Pathogenesis, Rna Interference, Translation, Transport
Quite a number of genes have been found to play important roles in Wolf-hirschhorn Syndrome, such as CDK2AP2, CDKN2A, CDKN2B, FGFR3, INVS, LAMTOR2, LETM1, LMLN, MRPL28, NXT1, PTER, RPP14, S100A9, SUB1, TMED10. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.