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Disease Info Card
Multiple Congenital Anomalies
Information about Multiple Congenital Anomalies: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Multiple Congenital Anomalies
Most recent studies have shown that Multiple Congenital Anomalies shares some biological mechanisms with atresia, cleft-lip, cleft-palate, congenital-abnormality, congenital-absence, congenital-heart-defects, cytogenetic-abnormality, developmental-delay-(disorder), dysplasia, embryonic-mosaic, growth-retardation, hypoplasia, microcephaly, micrognathism, muscle-hypotonia, orbital-separation-excessive, partial-trisomy, trisomy.
Among the many pathways, these few ones have gauged particular interests from scientists studying Multiple Congenital Anomalies, and have been seen in publications frequently: Bone Maturation, Cell Migration, Chromosome Breakage, Dna Repair, Fertilization, Hypersensitivity, Innervation, Interphase, Localization, Meiosis, Meiosis I, Metaphase, Myelination, Ossification, Pathogenesis, Pigmentation, Reflex, Segmentation, Transport, Transposition
Quite a number of genes have been found to play important roles in Multiple Congenital Anomalies, such as CDK5R1, CDKN2B, CHP1, DHCR7, ENDOU, EXOSC6, INS, INVS, LMLN, MB, MRPL28, NXT1, RAI1, S100A10, SUB1, TBX1, ZEB2. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.