Disease Info Card

Staphylococcus Aureus Infection

Information about Staphylococcus Aureus Infection: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Staphylococcus Aureus Infection

Most recent studies have shown that Staphylococcus Aureus Infection shares some biological mechanisms with abscess, arthritis, arthritis-infectious, bacteremia, bacterial-endocarditis, bacterial-infections, community-acquired-infections, cross-infection, endocarditis, infective-disorder, inflammation, mrsa---methicillin-resistant-staphylococcus-aureus-infection, osteomyelitis, pneumonia, septicemia, staphylococcal-infections, staphylococcal-skin-infections, surgical-wound-infection, systemic-infection, wound-infection.

Among the many pathways, these few ones have gauged particular interests from scientists studying Staphylococcus Aureus Infection, and have been seen in publications frequently: Autolysis, Biofilm Formation, Bioluminescence, Cell Activation, Cell Death, Cell Proliferation, Coagulation, Drug Resistance, Excretion, Immune Response, Inflammatory Response, Innate Immune Response, Lactation, Localization, Pathogenesis, Phagocytosis, Secretion, Transport, Virulence, Wound Healing

Quite a number of genes have been found to play important roles in Staphylococcus Aureus Infection, such as ALB, CRP, CSRP1, DEFB4A, FN1, IFNG, IL10, IL1B, IL2, IL4, IL6, MPO, NOS2, SLC9A6, TLR2, TLR4, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.