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Disease Info Card
Sinus Node Dysfunction (disorder)
Information about Sinus Node Dysfunction (disorder): characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Sinus Node Dysfunction (disorder)
Most recent studies have shown that Sinus Node Dysfunction (disorder) shares some biological mechanisms with atrial-fibrillation, atrial-flutter, atrioventricular-block, bradycardia, cardiac-arrhythmia, cardiac-fibrillation, cardiomyopathies, complete-atrioventricular-block, congenital-heart-defects, heart-block, heart-diseases, heart-failure, sick-sinus-syndrome, sinoatrial-block, sinus-arrest, supraventricular-tachycardia, syncope, tachycardia-ventricular.
Among the many pathways, these few ones have gauged particular interests from scientists studying Sinus Node Dysfunction (disorder), and have been seen in publications frequently: Aging, Cardiac Conduction, Cell Death, Cellular Localization, Circadian Rhythm, Hypersensitivity, Innervation, Localization, Membrane Depolarization, Membrane Organization, Pathogenesis, Protein Transport, Reflex, Regulation Of Heart Rate, Response To Activity, Senescence, Translation, Transport, Transposition, Vasodilation
Quite a number of genes have been found to play important roles in Sinus Node Dysfunction (disorder), such as AMY2A, BLOC1S6, GJA1, GNL3, HCN4, KRAS, KRT5, KRT7, PGR, PRB1, PRH1, SCG2, SCLY, SCN5A, SIGLEC1, TAL1, TMEM37. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.