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Disease Info Card
Radial Polydactyly
Information about Radial Polydactyly: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Radial Polydactyly
Most recent studies have shown that Radial Polydactyly shares some biological mechanisms with aplasia-nos, cleft-lip, cleft-palate, congenital-abnormality, congenital-absence, congenital-foot-deformity, congenital-hand-deformities, craniosynostosis, dysplasia, greig-cephalopolysyndactyly-syndrome, hernia, hypoplasia, limb-deformities-congenital, microcephaly, polydactyly, polysyndactyly, syndactyly.
Among the many pathways, these few ones have gauged particular interests from scientists studying Radial Polydactyly, and have been seen in publications frequently: Brain Development, Cell Adhesion, Cell Cycle, Cell Death, Cell Fate Determination, Cell Proliferation, Eye Development, Gastrulation, Hypersensitivity, Interphase, Limb Development, Limb Morphogenesis, Localization, Ossification, Pathogenesis, Programmed Cell Death, Segmentation, Spinal Cord Development, Tissue Development, Translation
Quite a number of genes have been found to play important roles in Radial Polydactyly, such as ALX4, BMP2, BMP4, CCDC6, F9, FGF4, FGF8, GLI1, GLI3, HAND2, HOXD13, HPD, LMBR1, RET, SALL1, SHH, VSX1, ZP2. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.