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Disease Info Card
Pyoderma Gangrenosum
Information about Pyoderma Gangrenosum: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Pyoderma Gangrenosum
Most recent studies have shown that Pyoderma Gangrenosum shares some biological mechanisms with arthritis, colitis, crohn-disease, dermatologic-disorders, erythema, gangrene, inflammation, inflammatory-bowel-diseases, intestinal-diseases, leg-ulcer, leukemia, malignant-neoplasms, pyoderma, rheumatoid-arthritis, skin-ulcer, sweet-syndrome, ulcer, ulcerative-colitis, vasculitis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Pyoderma Gangrenosum, and have been seen in publications frequently: Acute Inflammatory Response, Angiogenesis, Cell Activation, Chemotaxis, Coagulation, Cytokine Production, Dehiscence, Hypersensitivity, Immune Response, Inflammatory Response, Innate Immune Response, Keratinocyte Proliferation, Leukocyte Chemotaxis, Localization, Neutrophil Chemotaxis, Pathogenesis, Phagocytosis, Pigmentation, Secretion, Wound Healing
Quite a number of genes have been found to play important roles in Pyoderma Gangrenosum, such as ACAD8, C3, CRP, CSF2, CSF3, MEFV, MPO, MVK, NLRP3, NOD2, PSTPIP1, SF3B1, TNF, VEGFA, YWHAE. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.