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- Table of Contents
Information about Proteostasis Deficiencies: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Proteostasis Deficiencies shares some biological mechanisms with alzheimers-disease, amyloidosis, amyotrophic-lateral-sclerosis, cystic-fibrosis, dementia, diabetes-mellitus, diseases-protein-folding, endoplasmic-reticulum-stress, huntington-disease, inflammation, malignant-neoplasms, nerve-degeneration, neurodegenerative-disorders, parkinson-disease, physiological-stress, primary-lateral-sclerosis, prion-diseases, retinitis-pigmentosa, sclerosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Proteostasis Deficiencies, and have been seen in publications frequently: Aging, Amyloid Fibril Formation, Autophagy, Cell Death, Cellular Homeostasis, Cellular Process, Immune Response, Inflammatory Response, Macroautophagy, Mitochondrial Fission, Pathogenesis, Programmed Cell Death, Protein Folding, Protein Maturation, Proteolysis, Rna Interference, Secondary Growth, Secretion, Secretory Pathway, Transport
Quite a number of genes have been found to play important roles in Proteostasis Deficiencies, such as ABCB6, AR, C4BPA, CARD14, CFTR, CRYAB, HSF1, HSPA4, HSPB1, HTT, IGFALS, INS, MAPT, PRDX2, PRNP, RHO, SLC6A4, SNCA, SOD1, VCP. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.