Disease Info Card

Endoplasmic Reticulum Stress

Information about Endoplasmic Reticulum Stress: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Endoplasmic Reticulum Stress

Most recent studies have shown that Endoplasmic Reticulum Stress shares some biological mechanisms with alzheimers-disease, atherosclerosis, carcinoma, diabetes-mellitus, diabetes-mellitus-non-insulin-dependent, fatty-liver, fibrosis, hypoxia, inflammation, insulin-resistance, ischemia, liver-carcinoma, liver-diseases, malignant-neoplasms, neoplasms, nerve-degeneration, neurodegenerative-disorders, obesity, physiological-stress, steatohepatitis.

Among the many pathways, these few ones have gauged particular interests from scientists studying Endoplasmic Reticulum Stress, and have been seen in publications frequently: Autophagy, Cell Cycle, Cell Death, Cell Growth, Cell Proliferation, Glycosylation, Immune Response, Induction Of Apoptosis, Inflammatory Response, Insulin Secretion, Localization, Neuroprotection, Pathogenesis, Programmed Cell Death, Protein Folding, Response To Endoplasmic Reticulum Stress, Rna Interference, Secretion, Translation, Transport

Quite a number of genes have been found to play important roles in Endoplasmic Reticulum Stress, such as AKT1, ATF4, ATF6, BAX, BCL2, CASP3, DDIT3, EIF2AK3, ERN1, GDF10, HSP90B1, HSPA5, INS, MAPK8, XBP1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.