Disease Info Card

Proliferative Vitreoretinopathy

Information about Proliferative Vitreoretinopathy: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Proliferative Vitreoretinopathy

Most recent studies have shown that Proliferative Vitreoretinopathy shares some biological mechanisms with cataract, diabetic-retinopathy, disorder-of-eye, epiretinal-membrane, eye-injuries, giving-way, glaucoma, hemorrhage, inflammation, intraocular-pressure-disorder, macular-hole, macule, pathologic-neovascularization, proliferative-diabetic-retinopathy, retinal-detachment, retinal-diseases, retinal-perforations, rhegmatogenous-retinal-detachment, uveitis, vitreous-hemorrhage.

Among the many pathways, these few ones have gauged particular interests from scientists studying Proliferative Vitreoretinopathy, and have been seen in publications frequently: Angiogenesis, Cell Adhesion, Cell Cycle, Cell Death, Cell Growth, Cell Migration, Cell Proliferation, Chemotaxis, Coagulation, Dedifferentiation, Enucleation, Epithelial Cell Proliferation, Fibroblast Proliferation, Induction Of Apoptosis, Inflammatory Response, Localization, Pathogenesis, Secretion, Transdifferentiation, Wound Healing

Quite a number of genes have been found to play important roles in Proliferative Vitreoretinopathy, such as EGF, ERMAP, FGF2, FN1, FZD4, GFAP, HNRNPC, IL6, MAPK1, MAPK3, PEX7, PHYH, PVR, RPE, TNF, VEGFA, VIM. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.