Disease Info Card

Rhegmatogenous Retinal Detachment

Information about Rhegmatogenous Retinal Detachment: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Rhegmatogenous Retinal Detachment

Most recent studies have shown that Rhegmatogenous Retinal Detachment shares some biological mechanisms with cataract, diabetic-retinopathy, disorder-of-eye, edema, epiretinal-membrane, giving-way, glaucoma, hemorrhage, intraocular-pressure-disorder, intraoperative-complications, macular-hole, macule, myopia, proliferative-diabetic-retinopathy, proliferative-vitreoretinopathy, retinal-detachment, retinal-diseases, retinal-perforations, vitreous-detachment, vitreous-hemorrhage.

Among the many pathways, these few ones have gauged particular interests from scientists studying Rhegmatogenous Retinal Detachment, and have been seen in publications frequently: Aging, Angiogenesis, Blood Coagulation, Cell Activation, Cell Adhesion, Cell Migration, Cell Proliferation, Coagulation, Dehiscence, Delamination, Enucleation, Immune Response, Inflammatory Response, Localization, Pathogenesis, Pigmentation, Reflex, Regeneration, Transport, Wound Healing

Quite a number of genes have been found to play important roles in Rhegmatogenous Retinal Detachment, such as COX5A, ERG, ERMAP, FN1, IL6, KCNH2, MMP2, PEX7, PHYH, PVR, RPE, SRF, TIMP1, TNF, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.