Disease Info Card

Porphyria Cutanea Tarda

Information about Porphyria Cutanea Tarda: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Porphyria Cutanea Tarda

Most recent studies have shown that Porphyria Cutanea Tarda shares some biological mechanisms with bacterial-infections, bulla, carcinoma, dermatologic-disorders, disorders-of-porphyrin-metabolism, hemochromatosis, hepatitis, hepatitis-c, infective-disorder, inflammation, inflammatory-response, iron-overload, liver-cirrhosis, liver-diseases, malignant-neoplasms, neoplasms, pneumonia, systemic-infection, systemic-inflammatory-response-syndrome, virus-diseases.

Among the many pathways, these few ones have gauged particular interests from scientists studying Porphyria Cutanea Tarda, and have been seen in publications frequently: Angiogenesis, Bicarbonate Transport, Cell Growth, Chloride Transport, Coagulation, Excretion, Glomerular Filtration, Hemostasis, Immune Response, Inflammatory Response, Insemination, Localization, Natriuresis, Pathogenesis, Pigmentation, Reverse Transcription, Secretion, Segmentation, Transport, Transposition

Quite a number of genes have been found to play important roles in Porphyria Cutanea Tarda, such as ALB, CALCA, CRP, CSRP1, DCT, FECH, HFE, HMBS, IL10, IL6, LBP, NHS, PTH, ST8SIA4, SULT1A1, TF, TNF, UROD. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.