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Disease Info Card
Paraproteinemias
Information about Paraproteinemias: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Paraproteinemias
Most recent studies have shown that Paraproteinemias shares some biological mechanisms with amyloidosis, blood-protein-disorders, cryoglobulinemia, dysplasia, kidney-diseases, leukemia, lymphoma, malignant-neoplasms, malignant-paraganglionic-neoplasm, monoclonal-gammapathies, monoclonal-gammopathy-of-undetermined-significance, multiple-myeloma, neoplasms, peripheral-neuropathy, plasma-cell-dyscrasia, plasmacytoma, polyneuropathy, waldenstrom-macroglobulinemia.
Among the many pathways, these few ones have gauged particular interests from scientists studying Paraproteinemias, and have been seen in publications frequently: Aging, Angiogenesis, Bone Resorption, Cell Adhesion, Cell Cycle, Cell Growth, Cell Proliferation, Coagulation, Complement Activation, Excretion, Hemostasis, Immune Response, Immunoglobulin Production, Interphase, Localization, Pathogenesis, Pigmentation, Platelet Aggregation, Secretion, Transport
Quite a number of genes have been found to play important roles in Paraproteinemias, such as ALB, C3, C4A, CD19, CD38, CRP, CTLA4, HLA-DQA1, IL6, KRT20, MAG, MS4A1, MYOM2, NCAM1, NOD2, SDC1, TNF, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.