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Disease Info Card
Plasma Cell Dyscrasia
Information about Plasma Cell Dyscrasia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Plasma Cell Dyscrasia
Most recent studies have shown that Plasma Cell Dyscrasia shares some biological mechanisms with amyloidosis, dysplasia, endocrine-system-diseases, kidney-diseases, leukemia, lymphoma, malignant-neoplasms, malignant-paraganglionic-neoplasm, monoclonal-gammapathies, monoclonal-gammopathy-of-undetermined-significance, multiple-myeloma, neoplasms, paraproteinemias, plasmacytoma, poems-syndrome, polyneuropathy, primary-amyloidosis, waldenstrom-macroglobulinemia.
Among the many pathways, these few ones have gauged particular interests from scientists studying Plasma Cell Dyscrasia, and have been seen in publications frequently: Angiogenesis, B Cell Differentiation, Bone Resorption, Cell Cycle, Cell Differentiation, Cell Growth, Cell Proliferation, Dna Repair, Excretion, Glomerular Filtration, Glycosylation, Immune Response, Interphase, Localization, Metaphase, Methylation, Pathogenesis, Pigmentation, Proteolysis, Secretion
Quite a number of genes have been found to play important roles in Plasma Cell Dyscrasia, such as ALB, CD38, CTLA4, DNAH5, DNAI1, HLA-DQA1, IL2, IL6, KRT20, MAG, MS4A1, MYC, MYOM2, NOD2, PCBD1, PKD2L1, SDC1, TNF, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.