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Disease Info Card
Oxygen Toxicity
Information about Oxygen Toxicity: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Oxygen Toxicity
Most recent studies have shown that Oxygen Toxicity shares some biological mechanisms with anoxia, bronchopulmonary-dysplasia, convulsions, dysbarism, dysplasia, edema, fibrosis, hypoxia, inflammation, lung-diseases, lung-injury, nervousness, pneumonia, poisoning, pulmonary-edema, respiratory-distress, respiratory-distress-syndrome-adult.
Among the many pathways, these few ones have gauged particular interests from scientists studying Oxygen Toxicity, and have been seen in publications frequently: Aging, Cell Death, Cell Division, Cell Growth, Cell Proliferation, Chemotaxis, Conjugation, Dna Repair, Glycolysis, Hypersensitivity, Inflammatory Response, Localization, Lung Development, Lung Growth, Pathogenesis, Photosynthesis, Protein Oxidation, Response To Hyperoxia, Transport, Vasoconstriction
Quite a number of genes have been found to play important roles in Oxygen Toxicity, such as ACE, ALB, ATM, C2, CAT, CRAT, EPO, G6PD, GLYAT, GSR, HMOX1, IL6, NOS1, NOS2, SOD1, SOD2, TNF, XDH. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.