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Information about Nicotinamide Adenine Dinucleotide Coenzyme Q Reductase Deficiency: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Nicotinamide Adenine Dinucleotide Coenzyme Q Reductase Deficiency shares some biological mechanisms with acidosis, acidosis-lactic, atrophy, cardiomyopathies, disorder-of-the-optic-nerve, encephalopathies, leigh-disease, melas-syndrome, mitochondrial-diseases, mitochondrial-encephalomyopathies, mitochondrial-myopathies, muscle-hypotonia, myopathy, nerve-degeneration, neurodegenerative-disorders, parkinson-disease, weakness.
Among the many pathways, these few ones have gauged particular interests from scientists studying Nicotinamide Adenine Dinucleotide Coenzyme Q Reductase Deficiency, and have been seen in publications frequently: Aging, Cell Activation, Cell Death, Electron Transport, Electron Transport Chain, Fatty Acid Oxidation, Localization, Mitochondrial Depolarization, Mitochondrial Fission, Mitochondrial Fusion, Mitochondrial Translation, Necrotic Cell Death, Neuroprotection, Oxidative Phosphorylation, Pathogenesis, Programmed Cell Death, Response To Water Deprivation, Translation, Transport, Tricarboxylic Acid Cycle
Quite a number of genes have been found to play important roles in Nicotinamide Adenine Dinucleotide Coenzyme Q Reductase Deficiency, such as ACAD9, CYCS, IVNS1ABP, MT-ND3, NDP, NDUFAF1, NDUFB6, NDUFS1, NDUFS2, NDUFS4, NDUFS7, NDUFS8, NDUFV1, POLG. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.