Disease Info Card

Meningitis Neonatal

Information about Meningitis Neonatal: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Meningitis Neonatal

Most recent studies have shown that Meningitis Neonatal shares some biological mechanisms with abscess, bacteremia, bacterial-infections, brain-abscess, cell-invasion, enterobacteriaceae-infections, escherichia-coli-infections, group-b-streptococcal-infection, hydrocephalus, infective-disorder, meningitis, meningitis-bacterial, meningitis-escherichia-coli, neurological-ventriculitis, pneumonia, septicemia, sequelae-aspects, streptococcal-infections, systemic-infection, urinary-tract-infection.

Among the many pathways, these few ones have gauged particular interests from scientists studying Meningitis Neonatal, and have been seen in publications frequently: Astrocyte Activation, Cell Adhesion, Drug Resistance, Fermentation, Habituation, Hormone Secretion, Immune Response, Inflammatory Response, Innate Immune Response, Localization, Pathogenesis, Phagocytosis, Phagosome Acidification, Reflex, Secretion, Secretory Pathway, Transcytosis, Transport, Tropism, Virulence

Quite a number of genes have been found to play important roles in Meningitis Neonatal, such as ASAP2, BBS9, C5, CRP, CSF2, KRT1, LAMC2, MRPS30, MS4A1, PAPOLA, PDAP1, PLEKHM1, PTK2, REG3A, SPANXB1, TNF, TUSC2. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.