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- Table of Contents
Information about Malaria, Cerebral: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Malaria, Cerebral shares some biological mechanisms with anemia, brain-diseases, comatose, convulsions, edema, hemorrhage, hypoglycemia, infective-disorder, inflammation, kidney-failure, malaria, malaria-falciparum, meningitis, nervousness, parasitemia, plasmodium-falciparum-infection, respiratory-distress, sequelae-aspects, tissue-adhesions.
Among the many pathways, these few ones have gauged particular interests from scientists studying Malaria, Cerebral, and have been seen in publications frequently: Antigenic Variation, Cell Activation, Cell Adhesion, Cell Death, Coagulation, Cognition, Complement Activation, Cytokine Production, Drug Resistance, Endothelial Cell Activation, Immune Response, Inflammatory Response, Innate Immune Response, Localization, Pathogenesis, Phagocytosis, Secretion, T Cell Activation, Transport, Virulence
Quite a number of genes have been found to play important roles in Malaria, Cerebral, such as CD36, CD4, CD8A, CSF2, CTLA4, EPO, HLA-DQA1, ICAM1, IFNG, IL10, IL2, IL4, IL6, LAMC2, NOD2, NOS2, SELE, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.