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Disease Info Card
Macular Corneal Dystrophy Type I
Information about Macular Corneal Dystrophy Type I: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Macular Corneal Dystrophy Type I
Most recent studies have shown that Macular Corneal Dystrophy Type I shares some biological mechanisms with angiolymphoid-hyperplasia, fatty-liver, fibrosis, focal-glomerulosclerosis, glomerulonephritis, glomerulonephritis-minimal-change, glomerulosclerosis-(disorder), inflammation, kaposi-sarcoma, kidney-diseases, lipoid-nephrosis, lymphoma, multicentric-angiofollicular-lymphoid-hyperplasia, neoplasms, nephrotic-syndrome, nonalcoholic-steatohepatitis, proteinuria-of-undiagnosed-cause, segmental-glomerulosclerosis, steatohepatitis, steatosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Macular Corneal Dystrophy Type I, and have been seen in publications frequently: Angiogenesis, Brain Development, Cell Activation, Cell Cycle, Cell Death, Cell Proliferation, Coagulation, Endocytosis, Excretion, Exocytosis, Fatty Acid Oxidation, Glomerular Filtration, Inflammatory Response, Localization, Mast Cell Degranulation, Pathogenesis, Regeneration, Secretion, Sulfation, Transport
Quite a number of genes have been found to play important roles in Macular Corneal Dystrophy Type I, such as ACTN4, ALB, CAT, CHST6, COL18A1, CTLA4, CYCS, IL6, INS, MB, MLYCD, NOD2, OXSM, PPARA, PPARG, SLC17A5, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.