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Disease Info Card
Lupus Nephritis
Information about Lupus Nephritis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Lupus Nephritis
Most recent studies have shown that Lupus Nephritis shares some biological mechanisms with arthritis, autoimmune-diseases, autoimmune-reaction, autoimmunity, glomerulonephritis, glomerulonephritis-membranoproliferative, glomerulosclerosis-(disorder), hypertensive-disease, iga-glomerulonephritis, inflammation, kidney-diseases, kidney-failure, kidney-failure-chronic, lupus-erythematosus-systemic, membranous-glomerulonephritis, nephritis, nephrotic-syndrome, proteinuria-of-undiagnosed-cause, vasculitis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Lupus Nephritis, and have been seen in publications frequently: B Cell Activation, Cell Activation, Cell Adhesion, Cell Death, Cell Proliferation, Coagulation, Complement Activation, Cytokine Production, Excretion, Glomerular Filtration, Hypersensitivity, Immune Complex Formation, Immune Response, Inflammatory Response, Localization, Lymphocyte Activation, Pathogenesis, Secretion, T Cell Activation, T Cell Proliferation
Quite a number of genes have been found to play important roles in Lupus Nephritis, such as ACE, ALB, C3, C4A, CCL2, CD4, CD40LG, CTLA4, HLA-DQA1, IFNG, IGHG3, IL10, IL2, IL4, IL6, NOD2, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.