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Disease Info Card
Linear Iga Bullous Dermatosis
Information about Linear Iga Bullous Dermatosis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Linear Iga Bullous Dermatosis
Most recent studies have shown that Linear Iga Bullous Dermatosis shares some biological mechanisms with autoimmune-diseases, autoimmune-reaction, bulla, bullous-dermatitis, bullous-pemphigoid, dermatitis, dermatitis-herpetiformis, dermatologic-disorders, drug-eruptions, epidermolysis-bullosa, epidermolysis-bullosa-acquisita, exanthema, lupus-erythematosus-systemic, mucinous-adenocarcinoma, pemphigoid-benign-mucous-membrane, pemphigoid-gestationis, skin-diseases-vesiculobullous, ulcer.
Among the many pathways, these few ones have gauged particular interests from scientists studying Linear Iga Bullous Dermatosis, and have been seen in publications frequently: Cell Adhesion, Cell-cell Adhesion, Cell-matrix Adhesion, Chemotaxis, Coagulation, Cytolysis, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Lymphocyte Proliferation, Neutrophil Chemotaxis, Pathogenesis, Proteolysis, Secretion, Sensitization, Transport
Quite a number of genes have been found to play important roles in Linear Iga Bullous Dermatosis, such as ASCC3, C3, COL17A1, CP, DLD, DSG1, DSG3, DST, IFNG, IGHA1, IL2, ITGB2, LAD1, NDUFA2, NOD2, PLEC, TNF, TNFRSF25. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.