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- Table of Contents
Information about Pemphigoid, Benign Mucous Membrane: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Pemphigoid, Benign Mucous Membrane shares some biological mechanisms with autoimmune-diseases, autoimmune-reaction, bulla, bullous-pemphigoid, cicatrix, conjunctival-diseases, conjunctivitis, corneal-diseases, dermatologic-disorders, disorder-of-eye, epidermolysis-bullosa, epidermolysis-bullosa-acquisita, lichen-planus, mouth-diseases, mucinous-adenocarcinoma, ocular-cicatricial-pemphigoid, skin-diseases-vesiculobullous.
Among the many pathways, these few ones have gauged particular interests from scientists studying Pemphigoid, Benign Mucous Membrane, and have been seen in publications frequently: Aging, Cell Adhesion, Cell Division, Cell Migration, Cell Proliferation, Cell-matrix Adhesion, Complement Activation, Fibroblast Proliferation, Glycosylation, Hypersensitivity, Immune Response, Keratinization, Localization, Pathogenesis, Protein Secretion, Proteolysis, Regeneration, Secretion, Tropism, Wound Healing
Quite a number of genes have been found to play important roles in Pemphigoid, Benign Mucous Membrane, such as AMT, C3, COL17A1, CP, CTLA4, DSG1, DSG3, DST, HLA-DQA1, HLA-DRB4, HSPG2, IL4, ITGB2, LAD1, NOD2, SERPINH1, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.