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Disease Info Card
Leri-weill Dyschondrosteosis
Information about Leri-weill Dyschondrosteosis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Leri-weill Dyschondrosteosis
Most recent studies have shown that Leri-weill Dyschondrosteosis shares some biological mechanisms with bone-diseases-developmental, congenital-abnormality, cytogenetic-abnormality, dislocations, dwarfism, dysplasia, growth-disorders, haploinsufficiency, hypertrophy, langer-mesomelic-dysplasia-syndrome, madelungs-deformity, osteochondrodysplasias, shwachman-syndrome, skeletal-dysplasia, turner-syndrome.
Among the many pathways, these few ones have gauged particular interests from scientists studying Leri-weill Dyschondrosteosis, and have been seen in publications frequently: Bone Development, Cell Proliferation, Chondrocyte Hypertrophy, Chondrocyte Proliferation, Endochondral Ossification, Hatching, Interphase, Limb Development, Localization, Meiosis, Meiosis I, Menarche, Ossification, Pathogenesis, Secretion, Segmentation, Spermatogenesis
Quite a number of genes have been found to play important roles in Leri-weill Dyschondrosteosis, such as CACNA1C, CHP1, DCTN3, DYNC1H1, F2R, FGFR3, GGH, GH1, NPPB, NR1I2, PHB2, SHOX, SHOX2, SLC52A2, SRY, TBXAS1, TYMS. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.