This website uses cookies to ensure you get the best experience on our website.
- Table of Contents
, No Gclids
Disease Info Card
Kallmann Syndrome
Information about Kallmann Syndrome: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Kallmann Syndrome
Most recent studies have shown that Kallmann Syndrome shares some biological mechanisms with amenorrhea, anosmia, congenital-abnormality, congenital-absence, cryptorchidism, delayed-puberty, dysplasia, hereditary-diseases, hypogonadism, hypogonadotropic-hypogonadism, hypoplasia, ichthyoses, idiopathic-hypogonadotropic-hypogonadism, infertility, nervousness, olfaction-disorders, pituitary-diseases, steroid-sulfatase-deficiency-disease.
Among the many pathways, these few ones have gauged particular interests from scientists studying Kallmann Syndrome, and have been seen in publications frequently: Aging, Axon Guidance, Cell Adhesion, Cell Migration, Cell Proliferation, Fertilization, Gonadotropin Secretion, Hormone Secretion, Immune Response, Innervation, Localization, Neurogenesis, Neuron Development, Neuron Migration, Olfactory Bulb Development, Ovulation, Pathogenesis, Secretion, Spermatogenesis, System Development
Quite a number of genes have been found to play important roles in Kallmann Syndrome, such as BRD2, CHD7, COL18A1, FGF8, FGFR1, GNRH1, GNRHR, IHH, KISS1R, NR0B1, OXSM, PLOD1, PRL, PROK2, PROKR2, SERPINA4, STS. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.