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- Table of Contents
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Disease Info Card
Intermittent Claudication
Information about Intermittent Claudication: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Intermittent Claudication
Most recent studies have shown that Intermittent Claudication shares some biological mechanisms with arterial-occlusive-diseases, arteriopathic-disease, arteriosclerosis, atherosclerosis, cardiovascular-diseases, claudication-(finding), coronary-heart-disease, diabetes-mellitus, gangrene, hypertensive-disease, ischemia, myocardial-infarction, pain, peripheral-arterial-diseases, peripheral-vascular-diseases, rest-pain, stenosis, thrombosis, vascular-diseases.
Among the many pathways, these few ones have gauged particular interests from scientists studying Intermittent Claudication, and have been seen in publications frequently: Aging, Angiogenesis, Blood Circulation, Coagulation, Excretion, Fibrinolysis, Hemostasis, Inflammatory Response, Localization, Neutrophil Activation, Pathogenesis, Platelet Activation, Platelet Aggregation, Reflex, Regeneration, Translation, Transport, Vasoconstriction, Vasodilation, Wound Healing
Quite a number of genes have been found to play important roles in Intermittent Claudication, such as ACD, ACE, ALB, BCHE, CELA3B, CLU, CRP, DHX40, ENOPH1, F11, IL6, INS, NDUFB6, PADI4, PTCRA, REST, SELP, VEGFA, VWF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.