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- Table of Contents
Information about Inflammatory Diarrhea: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Inflammatory Diarrhea shares some biological mechanisms with bacterial-infections, campylobacter-infection, cell-invasion, cholera, chronic-diarrhea, colitis, diarrhea, dysentery, dysentery-bacillary, enteritis, enterocolitis-pseudomembranous, escherichia-coli-infections, gastroenteritis, infective-disorder, inflammation, inflammatory-bowel-diseases, intestinal-diseases, pain, salmonella-infections, shigella-infections.
Among the many pathways, these few ones have gauged particular interests from scientists studying Inflammatory Diarrhea, and have been seen in publications frequently: Acute Inflammatory Response, Antiport, Cell Activation, Cell Cycle, Cell Death, Cytokine Production, Immune Response, Inflammatory Response, Innate Immune Response, Localization, Mast Cell Activation, Pathogenesis, Phagocytosis, Response To Toxin, Secretion, Sensitization, Transcytosis, Transepithelial Chloride Transport, Transport, Virulence
Quite a number of genes have been found to play important roles in Inflammatory Diarrhea, such as CASP1, CLDN1, CTLA4, EPHB2, LEP, LTA, MAPK1, MPO, OCLN, SPI1, TAC1, TLR5, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.