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Disease Info Card
Infection By Trichuris Trichiura
Information about Infection By Trichuris Trichiura: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Infection By Trichuris Trichiura
Most recent studies have shown that Infection By Trichuris Trichiura shares some biological mechanisms with amebiasis, ancylostomiasis, anemia, ascariasis, diarrhea, giardiasis, helminthiasis, hookworm-infections, hymenolepiasis, infective-disorder, intestinal-diseases-parasitic, nematode-infections, parasitic-diseases, schistosomiasis, strongyloidiasis, swine-diseases, teniasis, trematode-infections, trichostrongyloidiasis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Infection By Trichuris Trichiura, and have been seen in publications frequently: Adaptive Immune Response, Cell Activation, Cell Differentiation, Cell Migration, Cell Proliferation, Cytokine Production, Defecation, Epithelial Cell Proliferation, Excretion, Flocculation, Humoral Immune Response, Hypersensitivity, Immune Response, Inflammatory Response, Lactation, Long-term Memory, Parasitism, Pathogenesis, Secretion, Type 2 Immune Response
Quite a number of genes have been found to play important roles in Infection By Trichuris Trichiura, such as ALB, CD4, CRP, CTLA4, ENPP3, HM13, IFNG, IGHE, IL10, IL13, IL18, IL4, IL5, IL6, IL9, ITGA2, STH, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.