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Disease Info Card
Idiopathic Hypereosinophilic Syndrome
Information about Idiopathic Hypereosinophilic Syndrome: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Idiopathic Hypereosinophilic Syndrome
Most recent studies have shown that Idiopathic Hypereosinophilic Syndrome shares some biological mechanisms with blood-coagulation-disorders, blood-loss-surgical, edema, eosinophilia, eosinophilic-leukemia, fibrosis, hemorrhage, hypereosinophilic-syndrome, hypotension-adverse-event, inflammation, leukemia, malignant-neoplasms, myeloproliferative-disease, neoplasms, nervousness, shock-hemorrhagic, systemic-infection, teratoma, thrombosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Idiopathic Hypereosinophilic Syndrome, and have been seen in publications frequently: Blood Coagulation, Cell Cycle, Cell Death, Cell Differentiation, Cell Growth, Cell Proliferation, Coagulation, Erythrocyte Aggregation, Excretion, Hemostasis, Inflammatory Response, Localization, Neurogenesis, Notch Signaling Pathway, Oxygen Transport, Pathogenesis, Platelet Aggregation, Regeneration, Secretion, Transport
Quite a number of genes have been found to play important roles in Idiopathic Hypereosinophilic Syndrome, such as ALB, F2, FGF2, FIP1L1, HES1, HES5, IL5, IL6, NANOGP8, NHS, NOTCH1, PDGFRA, POU5F1, RRBP1, SGSM3, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.