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Disease Info Card
Hypozincaemia
Information about Hypozincaemia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Hypozincaemia
Most recent studies have shown that Hypozincaemia shares some biological mechanisms with acrodermatitis-enteropathica, acute-phase-reaction, copper-deficiency, depressive-disorder, dermatitis, diabetes-mellitus, diarrhea, heart-failure, hypercupremia, hyperparathyroidism-secondary, hypocalcemia, inflammation, kidney-failure, kidney-failure-chronic, leukocytosis, malignant-neoplasms, malnutrition, zinc-deficiency-disorder.
Among the many pathways, these few ones have gauged particular interests from scientists studying Hypozincaemia, and have been seen in publications frequently: Acute-phase Response, Cell Death, Diuresis, Excretion, Gluconeogenesis, Glucose Homeostasis, Hypersensitivity, Immune Response, Inflammatory Response, Intestinal Absorption, Lactation, Menstruation, Neurogenesis, Pathogenesis, Platelet Aggregation, Secretion, Transport, Urea Cycle, Vitamin A Transport, Wound Healing
Quite a number of genes have been found to play important roles in Hypozincaemia, such as ACE, ALB, C1QL1, CP, CRH, CRP, DBI, EPO, HAMP, IL1B, IL6, INS, MCAT, POMC, PRL, PTH, PTRH1, SLC39A14, TF, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.