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Disease Info Card
Acrodermatitis Enteropathica
Information about Acrodermatitis Enteropathica: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Acrodermatitis Enteropathica
Most recent studies have shown that Acrodermatitis Enteropathica shares some biological mechanisms with celiac-disease, crohn-disease, deficiency-diseases, dermatitis, dermatologic-disorders, diarrhea, erythema, exanthema, gastrointestinal-diseases, hereditary-diseases, hypozincaemia, immunologic-deficiency-syndromes, infantile-diarrhea, infective-disorder, intestinal-diseases, malabsorption-syndrome, malnutrition, paronychia-inflammation, zinc-deficiency-disorder.
Among the many pathways, these few ones have gauged particular interests from scientists studying Acrodermatitis Enteropathica, and have been seen in publications frequently: Aging, Cell Death, Cell Division, Chemotaxis, Endocytosis, Excretion, Hypersensitivity, Intestinal Absorption, Keratinization, Localization, Lymphocyte Proliferation, Monocyte Chemotaxis, Pathogenesis, Pigmentation, Programmed Cell Death, Prolactin Secretion, Secretion, Telogen, Transport, Wound Healing
Quite a number of genes have been found to play important roles in Acrodermatitis Enteropathica, such as ALB, AMY2A, BLOC1S6, CALM3, DAPK3, GLB1, IL2, LCT, LYZ, NT5E, PRL, SLC30A1, SLC30A4, SLC30A5, SLC39A4, SLC39A5, ZGPAT. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.