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Disease Info Card
Hyperuricaemic Nephropathy
Information about Hyperuricaemic Nephropathy: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Hyperuricaemic Nephropathy
Most recent studies have shown that Hyperuricaemic Nephropathy shares some biological mechanisms with arthritis, arthritis-gouty, chronic-kidney-disease, cystic-kidney-diseases, familial-juvenile-gout, fibrosis, gout, hypertensive-disease, hyperuricemia, kidney-diseases, kidney-failure, kidney-failure-chronic, medullary-cystic-disease, medullary-cystic-kidney-disease-type-2, nephritis, nephritis-interstitial, polycystic-kidney-autosomal-dominant, renal-insufficiency, tubulointerstitial-nephritis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Hyperuricaemic Nephropathy, and have been seen in publications frequently: Cell Cycle, Chemotaxis, Cytolysis, Diuresis, Endocytosis, Excretion, Glomerular Filtration, Glycosylation, Hypersensitivity, Ion Transport, Localization, Menopause, Pathogenesis, Protein Maturation, Protein Processing, Protein Transport, Receptor-mediated Endocytosis, Secretion, Secretory Pathway, Transport
Quite a number of genes have been found to play important roles in Hyperuricaemic Nephropathy, such as EGF, GLI2, GNPDA1, GPI, HNF1B, HPRT1, KCNJ1, KIF3A, MUC1, NPHP1, NPHP4, OAT, OIT3, RAPGEF5, REN, SLC22A12, UMOD. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.